In fear memory protocols, new threatening information undergoes a progressive stabilization process termed consolidation which requires de novo protein synthesis. This process converts the initial fragile trace into an established long-term memory . Upon retrieval and under certain boundary conditions, such memory trace can re-enter a transient labile state which requires a novel stabilization process dependent upon new protein synthesis referred to as reconsolidation. This process is not a faithful recapitulation of consolidation. In fact, non-overlapping and distinctive mechanisms between consolidation and reconsolidation have been described in selected brain regions. We demonstrated the interaction between a stressfulevent and the retrieval phase of a consolidated trace.Such interaction is dependent on BDNF mechanism and not in molecular events that characterize the reconsolidation mechanism. These findings suggest that such interaction is dependent on BDNF consolidation-like mechanism.