Resumen:
t is well recognized that stressful experiences promote robust emotional memories, which are well remembered. The amygdaloid
complex, principally the basolateral complex (BLA), plays a pivotal role in fear memory and in the modulation of stress-induced
emotional responses. A large number of reports have revealed that GABAergic interneurons provide a powerful inhibitory control of the activity of projecting glutamatergic neurons in the BLA. Indeed, a reduced GABAergic control in the BLA is essential
for the stress-induced influence on the emergence of associative fear memory and on the generation of long-term potentiation (LTP) in BLA neurons. The extracellular signal-regulated kinase (ERK) subfamily of the mitogen-activated protein kinase
(MAPK) signaling pathway in the BLA plays a central role in the consolidation process and synaptic plasticity. In support of
the view that stress facilitates long-term fear memory, stressed animals exhibited a phospho-ERK2 (pERK2) increase