Resumen:
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The liver constitutes the first barrier in the control of hematogenous dissemination of Candida
albicans of intestinal origin. In rats infected with C. albicans, this organ limits the growth of the yeast
and mounts an efficient inflammatory reaction. However, in rats infected and exposed to chronic
varied stress, the hepatic inflammatory reaction is compromised and the outcoming of the infection is
more severe. Although in both groups the fungal burden is associated with hepatotoxicity, steatosis,
increment of hepatic enzymes and lipid peroxidation, stress-related differences are clearly evident.
Herein, we evaluated in infected and infected-stressed hosts the involvement